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Nematodes suppress the immunity generated by infection as well as impact responses to other non-nematode antigens [1]. Some research have shown that autoimmune diseases are increasing in prevalence in areas where exposure to helminths is uncommon. These observations recommend that the loss of pathogens and parasites removes a organic governor that assists to stop illness due to immune regulation [2]. Epidemiological and laboratory studies confirm that nematodes prevent immunemediated ailments. The immunological mechanism underlying the nearby therapeutic impact of gastrointestinal nematodes on inflammatory bowel illnesses and on various inflammatory tissue will not be clearly understood and is currently becoming intensively investigated. It was previously recommended thatproteins released from nematodes suppress activation of the Th1 inflammatory response in the inflammatory tissue not just by way of modulation from the Th2 response but in addition by mechanisms dependent on macrophag.